Butterfield, D., Howard, B., & LaFontaine, M. (2001). Brain oxidative stress in animal models of accelerated aging and the age-related neurodegenerative disorders, Alzheimer’s disease and Huntington’s disease. Current Medicinal Chemistry, 8(7), 815-28. https://doi.org/10.2174/0929867013373048

Butterfield, D., Howard, B., Yatin, S., Allen, K., & Carney, J. (1997). Free radical oxidation of brain proteins in accelerated senescence and its modulation by N-tert-butyl-alpha-phenylnitrone. Proceedings of the National Academy of Sciences of the United States of America, 94(2), 674-8. https://doi.org/10.1073/pnas.94.2.674 (Original work published 1997)

Howard, B., Yatin, S., Hensley, K., Allen, K., Kelly, J., Carney, J., & Butterfield, D. (1996). Prevention of hyperoxia-induced alterations in synaptosomal membrane-associated proteins by N-tert-butyl-alpha-phenylnitrone and 4-hydroxy-2,2,6,6-tetramethylpiperidin-1-oxyl (Tempol). Journal of Neurochemistry, 67(5), 2045-50. https://doi.org/10.1046/j.1471-4159.1996.67052045.x

Harris, M., Carney, J., Cole, P., Hensley, K., Howard, B., Martin, L., … Butterfield, D. (1995). beta-Amyloid peptide-derived, oxygen-dependent free radicals inhibit glutamate uptake in cultured astrocytes: implications for Alzheimer’s disease. Neuroreport, 6(14), 1875-9. https://doi.org/10.1097/00001756-199510020-00013 (Original work published 1995)

Hensley, K., Howard, B., Carney, J., & Butterfield, D. (1995). Membrane protein alterations in rodent erythrocytes and synaptosomes due to aging and hyperoxia. Biochimica et Biophysica Acta, 1270(2-3), 203-6. https://doi.org/10.1016/0925-4439(95)00043-4 (Original work published 1995)