Swiatkowski, P., Sewell, E., Sweet, E., Dickson, S., Swanson, R., McEwan, S., … Firestein, B. (2018). Cypin: A novel target for traumatic brain injury. Neurobiology of Disease, 119, 13-25. https://doi.org/10.1016/j.nbd.2018.07.019 (Original work published 2018)

Liang, C., Carrel, D., Omelchenko, A., Kim, H., Patel, A., Fanget, I., & Firestein, B. (2018). Cortical Neuron Migration and Dendrite Morphology are Regulated by Carboxypeptidase E. Cerebral Cortex (New York, N.Y. : 1991). https://doi.org/10.1093/cercor/bhy155 (Original work published 2018)

Svane, K., Asis, E., Omelchenko, A., Kunnath, A., Brzustowicz, L., Silverstein, S., & Firestein, B. (2018). d-Serine administration affects nitric oxide synthase 1 adaptor protein and DISC1 expression in sex-specific manner. Molecular and Cellular Neurosciences, 89, 20-32. https://doi.org/10.1016/j.mcn.2018.03.011

Patel, M., Swiatkowski, P., Kwon, M., Rodriguez, A., Campagno, K., & Firestein, B. (2018). A Novel Short Isoform of Cytosolic PSD-95 Interactor (Cypin) Regulates Neuronal Development. Molecular Neurobiology, 55(8), 6269-6281. https://doi.org/10.1007/s12035-017-0849-z

Rodriguez, A., O’Neill, K., Swiatkowski, P., Patel, M., & Firestein, B. (2018). Overexpression of cypin alters dendrite morphology, single neuron activity, and network properties via distinct mechanisms. Journal of Neural Engineering, 15(1), 016020. https://doi.org/10.1088/1741-2552/aa976a

Kung, F., Sillitti, D., Shreiber, D., Zahn, J., & Firestein, B. (2018). Microfluidic device-assisted etching of p-HEMA for cell or protein patterning. Biotechnology Progress, 34(1), 243-248. https://doi.org/10.1002/btpr.2576

O’Neill, K., Kwon, M., Donohue, K., & Firestein, B. (2017). Distinct effects on the dendritic arbor occur by microbead versus bath administration of brain-derived neurotrophic factor. Cellular and Molecular Life Sciences : CMLS, 74(23), 4369-4385. https://doi.org/10.1007/s00018-017-2589-7

Swiatkowski, P., Nikolaeva, I., Kumar, G., Zucco, A., Akum, B., Patel, M., … Firestein, B. (2017). Role of Akt-independent mTORC1 and GSK3β signaling in sublethal NMDA-induced injury and the recovery of neuronal electrophysiology and survival. Scientific Reports, 7(1), 1539. https://doi.org/10.1038/s41598-017-01826-w (Original work published 2017)